APSGN remains a significant clinical entity in spite of declining incidence rate in the pediatric population in well- developed countries. In developed countries APSGN became very rare but may still appear in adults with comorbidities, primarily diabetes mellitus and morbid obesity. APSGN is still prevalent in many parts of the world. In the past, it was the most common and the most studied form of acute postinfectious glomerulonephritis. These extensive studies provided us with invaluable information about the pathogenesis of acute glomerulonephritis, not only APSGN, but other forms of glomerulonephritides as well. Acute glomerulonephritis has been known to follow certain infections a long time ago. Already, Hippocrates described the occurrence of back pain and gross hematuria leading to oliguria or anuria more than two millennia ago . About two centuries ago, Wells noted bloody urine in patients with scarlet fever and postscarlatinal anasarca . Later, Bright noted the association with scarlatina and described the finding of blood in the urine and swelling of the face in what were probably attacks of APSGN . Therefore, acute glomerulonephritis was named after Bright (Bright’s disease).